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Consequently, the continuous excavation of genes upstream and downstream of glutamine metabolism, which can influence cancer cell invasion and metastasis-related genes, will be a promising means to contribute to the design of clinically specific relevant inhibitors

Because folate deficiency may predispose to hyperhomocysteinemia in heterozygotes for the CBS defect, transsulfuration defects might be combined with remethylation defects in patients with basal hyperhomocysteinemia (Lentz et al, 2000)

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Appears to calm neuroinflammation as well, giving it potential in brain injury or neurological recovery

Cochrane Database of Systematic Reviews 2011.10 (2011): CD006423